What is Diabetic Foot?
The clinical condition that occurs with foot wounds caused by excessive pressure on the basis of ischemia (reduced blood flow) as a result of nerve damage and occlusive vascular disease that occur as complications of diabetes, and the infection added to it, is called diabetic foot. One fifth of diabetic patients do not have to stand at some point in their lives. wound, ulcer or infection develops. In particular, diabetic foot accounts for 20 percent of the reasons why patients apply to the hospital. In addition, 50-70 percent of amputation surgeries are performed by patients with diabetes.
Causes of Diabetic Foot
There are two important causes of diabetic foot wounds: vascular occlusion and nerve damage. The organ most affected by nerve damage and occlusive vascular disease, which occur as complications of diabetes, is the patient's lower extremity.
Neuropathy is the leading cause of diabetic foot lesion onset. It has been understood that in most of the hospitalized patients, a physical factor that the patients are not aware of, such as hitting, hitting, stinging, burning, etc., initiated the wound. Patients often fail to recognize the injury at an early stage due to sensory loss and continue to stand and walk. This leads to progressive tissue damage.
The most important factor determining the outcome of diabetic foot ulcers, which are classified as neuropathic, ischemic and neuro-ischemic, is peripheral artery disease. Vascular damage in diabetic foot ulcers may occur in the form of macro and micro vessel involvement.
Common Risk Factors for Diabetic Foot
Common Risk Factors Description
Peripheral Neuropathy: Numbness/insensitivity to sensory stimuli (such as cold, tingling, burning, touch) in the patient's foot. Pain may occur starting from the fingertips and radiating towards the beginning of the limb.
Peripheral Artery Disease Peripheral artery disease is defined as the blockage or narrowing of the arteries in the leg area. Individuals with diabetes are four times more likely to develop peripheral artery disease than those without diabetes. This rate increases depending on the duration of diabetes and age factors.
Infection: As a result of infection in diabetic foot wounds, symptoms of edema, bad odor and pus are observed. However, individuals may not feel the necrosis occurring in the skin due to numbness (neuropathy). Infection-based pain and temperature increases may not be felt due to neuropathy
Duration of Diabetes: Another risk factor that is effective in the formation of diabetic foot wounds is the duration of diabetes.
Presence of Other Complications Due to Diabetes The presence of other complications related to diabetes is also effective in the formation of diabetic foot wounds.
Appropriate Shoe Selection The pressure applied to the bone protrusions in neuropathic feet with sensory loss may cause skin problems. As a result of choosing inappropriate shoes, the pressure applied to the area increases and this may cause skin ulcers.
Gender: Higher foot pressure in men compared to women causes a higher risk of ulcer formation in men.
Smoking is an independent risk factor for diabetic foot sores.
Not Paying Attention to Foot Care Health Paying attention to foot care and performing it regularly is very important in preventing diabetic foot wounds.
Uncontrolled Blood Sugar Level Another reason for the development of diabetic foot sores is the lack of blood sugar level control.
Trauma Due to the formation of neuropathy, patients cannot realize small or large traumas. Repetitive traumas are a risk factor that may cause diabetic foot wounds, in other words ulcers, to become chronic.
As the diabetic foot infection progresses, it spreads to the bloodstream and causes a condition called sepsis, which can be life-threatening.
Diabetic Foot Stages
In diabetic patients, the dryness of the feet caused by the impairment of the sweating mechanism due to autonomic neuropathy causes cracks, crevices and calluses on the skin. These cracks and crevices are entry points for fungi and other infectious agents. Infection causes cracks to grow and deepen. As a result of diabetes-related damage to sensory nerves (diabetic sensory neuropathy), the patient does not feel the infected wound and pain on his foot. As the wound grows and the infection increases, and by the time the patient becomes aware of the wound with the discharge, the wound has already become threatening to the foot and leg. As a result of vascular damage due to diabetes, wound healing is delayed due to insufficient blood supply to the foot.
There are stages of diabetic wound. Diabetic foot stages are listed as follows according to the Wagner Classification:
Stage 0: Healthy skin
Stage 1: Superficial ulcer
Stage 2: Deep ulcer
Stage 3: Ulcer with bone involvement
Stage 4: Forefoot (fingers/toe) gangrene
Stage 5: Gangrene of the entire foot
1.Examination of peripheral vascular structure,
2.Presence of neuropathy
3.The spread of the existing wound and infection in the bone and soft tissue, especially bone tissue inflammation (osteomyelitis), should be questioned.
4.Detection of the microorganism causing the infection (there may be more than one factor) by appropriate isolation methods and initiation of antibiotic therapy
5.Wound care and dressing
6. Wound closure and reconstruction procedures
Although problems can be seen in larger vessels in almost all patients with diabetic foot wounds, stenosis and blockages are definitely encountered in the small veins below the knee and in the feet. The vein that feeds the leg in the area below the knee divides into three and moves to the foot, and two of these veins unite in the foot to form an arch and network that feeds the entire foot and fingers. For wound healing, the treatments must provide blood supply to at least one vein that feeds the foot and fingers.
As a result of interventions made through needle holes in the groin and/or foot veins with closed methods called endovascular, foot nutrition can be provided in the angiography room without any incision.
Orthopedic Surgical Approach
Debridement is planned after evaluating the depth of the wound, if infection is present, which compartments it has spread to, and bone tissue involvement (osteomyelitis) using both physical examination and radiological imaging methods. The indispensable and most important part of wound treatment is debridement.
Debridement: It is the name given to the process of cleaning dead, dirty, infected tissues contaminated with microbes.
Debridement Methods:
Surgical (Sharp) Debridement
Larval Therapy
Enzymatic Debridement
Autolytic Debridement
Enzymatic Debridement
It can be achieved using a variety of enzymatic agents, including collagenase, papain, a combination of streptokinase and streptodornase, and dextrans.
Autolytic Debridement
It involves the use of dressings that maintain the moisture balance of the wound environment.
Thus, defense mechanisms (neutrophils, macrophages) can clean the tissues that have lost their vitality by using the body's enzymes.
Larval Therapy
It is a method in which fly larvae, known as green bottle flies, are used for biological debridement.
Antibiotic Treatment
Foot infections in diabetic patients require carefully selected antibiotic therapy in addition to debridement and wound care. Antibiotic selection is made according to the type of wound and laboratory results of the sample taken from the injured tissue. Treatment of diabetic foot infections should be started as soon as possible because the infection in the soft tissue can quickly spread to the deep tissues, making the treatment more difficult. Treatment duration varies from one week (for mild soft tissue infections) to 6 weeks (for osteomyelitis). Antibiotic treatment should be continued until the symptoms of infection improve.
Wound Care and Dressing
Diabetic foot wounds should be evaluated separately. The need for debridement should be evaluated for each dressing and dead tissue should be removed when necessary. First of all, during dressing, the wound should be washed with isotonic solution and the wound should be covered with sterile gases to keep it moist. The wound should not be wrapped too tightly and movement of the wound area should be restricted. There are different wound dressings that can be used depending on the current condition of the wound. As the condition of the wound changes, the dressing used may also change.
Wound Dressing Types
1.FILM COVER, TRANSPARENT FILM: The simplest covering, polyurethane structure, semi-permeable, protective for moisture balance. 1. On clean, exudate-free wounds awaiting epithelialization 2. On surgical incisions 3. On other products
2.ABSORBENT COVERS:Basic covers are used to absorb medium and large amounts of exudate. 1. HYDROCOLLOIDS 2. FIBER/ALGINATES 3. FOAMS
3. ABSORBING (EXUDATE ABSORBING PRODUCTS) Hydrocolloids: They are mostly in cellulose structure. They consist of substances such as Gelatin, Pectin and CMC.
4.GELS, HYDROGELS: Provides moisture balance in the wound and helps autolytic debridement.
5.ANTIBACTERIAL, ANTIMICROBIAL COVERS:
Wound Closure and Reconstruction
In wounds whose infection has been treated and sufficient clean granulation tissue has developed, the existing wound is surgically reconstructed with partial skin grafting, local flaps or free flap surgical methods.
The following should be taken into consideration to recognize the early "WARNING" symptoms of foot problems:
FOOT CARE